Discussion I believe that in the SN protocol, the longer we remain conscious after drinking SN, the higher our success rate of CTB will be. Isn't it？

[Zhoutongwei]

Let's take the use of antacids as an example:

If antacids are used → the production of nitric oxide is reduced → the drop in blood pressure caused by vasodilation occurs more slowly → this means it will take longer for us to lose consciousness → we are more likely to vomit while still conscious (and thus able to drink a second dose), rather than vomiting after losing consciousness (in which case we wouldn't know how much was vomited, nor would we be able to take a second dose of SN).

From this perspective, antacids are indeed very useful. Their purpose is not only to reduce the waste caused by SN reacting with stomach acid, but I believe their greatest significance is to delay the time we lose consciousness, thus ensuring that we are still awake when vomiting.

This debate is similar to whether the dosage of benzodiazepines should be, as stated in PPH, "enough to knock you out quickly" — I think rapidly entering a comatose state is unwise for a simple reason: if we vomit while unconscious, how can we drink a second dose?

What is your opinion？You're welcome to discuss.

 

[2messdup]

Let's take the use of antacids as an example:

If antacids are used → the production of nitric oxide is reduced → the drop in blood pressure caused by vasodilation occurs more slowly → this means it will take longer for us to lose consciousness → we are more likely to vomit while still conscious (and thus able to drink a second dose), rather than vomiting after losing consciousness (in which case we wouldn't know how much was vomited, nor would we be able to take a second dose of SN).

From this perspective, antacids are indeed very useful. Their purpose is not only to reduce the waste caused by SN reacting with stomach acid, but I believe their greatest significance is to delay the time we lose consciousness, thus ensuring that we are still awake when vomiting.

This debate is similar to whether the dosage of benzodiazepines should be, as stated in PPH, "enough to knock you out quickly" — I think rapidly entering a comatose state is unwise for a simple reason: if we vomit while unconscious, how can we drink a second dose?

What is your opinion？You're welcome to discuss.

As I understand it, 25g is more than enough to kill you. People have died after much less. The second drink is belt and braces but as long as you don't vomit all of the initial amount immediately you still have a good chance of dying with some vomiting of the first drink. Personally I'm avoiding antacids as I want as little as possible in my stomach but using benzos to reduce anxiety. Not going to aim for benzos to knock me out tho as I'm going to do the two drinks just to be sure.

 

[Zhoutongwei]

As I understand it, 25g is more than enough to kill you. People have died after much less. The second drink is belt and braces but as long as you don't vomit all of the initial amount immediately you still have a good chance of dying with some vomiting of the first drink. Personally I'm avoiding antacids as I want as little as possible in my stomach but using benzos to reduce anxiety. Not going to aim for benzos to knock me out tho as I'm going to do the two drinks just to be sure.

Regarding benzodiazepines, we share the same stance — we take them to reduce anxiety, absolutely not to hasten unconsciousness.

Once we fall unconscious, we can no longer know whether we will vomit afterward, let alone take a second dose.
Although many sources claim that even if vomiting occurs after the first dose, the amount of SN already absorbed by the body is sufficient to be lethal, I still believe there is a risk — because you don't truly know how much has been absorbed, or whether it's enough to cause death.
I don't want to gamble on probability. That's why I want to stay conscious for as long as possible, to be able to deal with vomiting if it happens.

 

[EmptyBottle]

I doubt that vomiting will be enough to both clear all the SN from the stomach, and clear enough of the absorbed SN from the bloodstream to make the hyper-lethal dose non-lethal. It may ofc slow the death down... but asphyxiating on stomach contents while unconscious may actually speed up the death